Difference between revisions of "Mdh"

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m (CitH moved to Mdh over redirect)
(Expression and regulation)
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=== Additional information===
 
=== Additional information===
  
=Expression and regulation=
+
* '''Operon:'''
 +
**''[[citZ]]-[[icd]]-[[mdh]]''
 +
**''[[icd]]-[[mdh]]''
  
* '''Operon:'''  
+
* '''Sigma factor:''' [[SigA]]
  
* '''Sigma factor:'''  
+
* '''Regulation:''' catabolite repression ([[CcpA]]), repression by glucose + glutamate ([[CcpC]])
 +
''[[mdh]]'': constitutive
 +
* '''Regulatory mechanism:''' [[CcpA]]: transcription repression, [[CcpC]]: transcription repression
  
* '''Regulation:'''
 
  
* '''Regulatory mechanism:'''
 
  
* '''Additional information:'''  
+
* '''Additional information:'''
  
 
=Biological materials =
 
=Biological materials =

Revision as of 15:57, 9 February 2009

  • Description: write here

Gene name mdh
Synonyms citH
Essential
Product malate dehydrogenase (EC 1.1.1.37)
Function
MW, pI 33 kDa, 4.727
Gene length, protein length 936 bp, 312 aa
Immediate neighbours
Gene sequence (+200bp) Protein sequence
Genetic context
Mdh context.gif



The gene

Basic information

  • Coordinates:

Phenotypes of a mutant

Database entries

  • DBTBS entry: [1]
  • SubtiList entry: [2]

Additional information

The protein

Basic information/ Evolution

  • Catalyzed reaction/ biological activity:
  • Protein family:
  • Paralogous protein(s):

Extended information on the protein

  • Kinetic information:
  • Domains:
  • Modification:
  • Cofactor(s):
  • Effectors of protein activity:
  • Interactions:
  • Localization:

Database entries

  • Structure:
  • Swiss prot entry:
  • KEGG entry:
  • E.C. number:

Additional information

  • Regulation: catabolite repression (CcpA), repression by glucose + glutamate (CcpC)

mdh: constitutive

  • Regulatory mechanism: CcpA: transcription repression, CcpC: transcription repression


  • Additional information:

Biological materials

  • Mutant:
  • Expression vector:
  • lacZ fusion:
  • GFP fusion:
  • two-hybrid system:
  • Antibody:

Labs working on this gene/protein

Your additional remarks

References

  1. Author1, Author2 & Author3 (year) Title Journal volume: page-page. PubMed